Adamax 10mg
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Adamax is a highly advanced, synthetic peptide analogue of Semax (an ACTH 4-10 fragment derivative). It is chemically engineered through two distinct structural modifications: the addition of an N-terminal acetyl group and a large, lipophilic C-terminal adamantane moiety.
In laboratory and research environments, this peptide is distinguished by its enhanced metabolic stability in sterile water and superior lipid-bilayer penetration compared to the base Semax peptide. Adamax serves as a premium reagent for investigating advanced neurotrophic signaling, cellular resilience, and peptide bioavailability mechanisms.
$105.00
In stock
In stock
Adamax 10MG Peptide
Research-Grade Enhanced Semax Analogue
Product Overview
Adamax is a highly advanced, synthetic peptide analogue of Semax (an ACTH 4-10 fragment derivative). It is chemically engineered through two distinct structural modifications: the addition of an N-terminal acetyl group and a large, lipophilic C-terminal adamantane moiety.
In laboratory and research environments, this peptide is distinguished by its enhanced metabolic stability in sterile water and superior lipid-bilayer penetration compared to the base Semax peptide. Adamax serves as a premium reagent for investigating advanced neurotrophic signaling, cellular resilience, and peptide bioavailability mechanisms.
Technical Specifications
| Sequence | Modified Heptapeptide |
|---|---|
| Molecular Formula | C₄₄H₆₁N₁₁O₁₃S |
| Structure Stability | Stable in sterile water |
| Analytical Purity | ≥99% (HPLC / MS Verified) |
| Format | Lyophilized Powder (10mg) |
Research Applications
Adamax is utilized in specialized laboratory models to investigate structural modifications and their biological implications:
- Neurotrophic Signaling: In vitro examination of Brain-Derived Neurotrophic Factor (BDNF) expression and related receptor sensitivities.
- Peptide Pharmacokinetics: Studies focusing on how bulky lipophilic additions (like adamantane) affect enzymatic degradation and half-life in physiological solvents.
- Cellular Stress Models: Observation of neuronal cell survival, structural protection, and functional preservation during induced oxidative or hypoxic stress.
Mechanism of Action
Like its parent peptide Semax, Adamax functions primarily by modulating the expression of neurotrophins and targeting the TrkB receptor pathways. However, the molecular mechanism is heavily influenced by the adamantane structural group, which significantly increases the compound’s lipophilicity.
This physical property allows Adamax to penetrate cellular membranes more efficiently in tissue cultures. In laboratory models, researchers observe that this enhanced bioavailability leads to prolonged receptor engagement, initiating sustained intracellular cascades that regulate neuroplasticity, memory consolidation correlates, and structural repair mechanisms without immediate degradation by serum proteases.
Important Safety Disclaimer
FOR RESEARCH AND LABORATORY USE ONLY.
Adamax is an experimental synthetic peptide. It is strictly not intended for human consumption, animal consumption, therapeutic application, or diagnostic procedures. This product must be handled by qualified professionals strictly adhering to laboratory safety protocols and governing regulations.
